Studies on Royal Jelly and Abnormal Cholesterol and Triglycerides
Fraction 1 contains the physiologically active material responsible for sexual development of the queen bees.

Some of the Studies:

Dr. G Townsend et al did extensive investigation on 10-Hydroxydecenoic acid in royal jelly on antitumor activity in mice. Acetylcholin is reported by William H. Brown (1959). Euphoria and strength after royal jelly consumption may be related to neurostimulating substance. A. Toman (1972), failed to demonstrate sex hormone increase in human male subjects.

Case Report on Royal Jelly Experiment for Abnormal Cholesterol & Triglyceride The following are experimental studies which were made by the author. The proper amount of royal jelly to be given to the patients was entirely unknown. The authors personal communication with Dr. Vasser, M.D. , at Miami University convinced me that large amounts of royal jelly are not harmful despite long term application in his geriatric studies.

Royal jelly was supplied by Willson Co. in New York (donated by Royal Jelly Fund). The royal jelly was bottled in colored plastic 1-pound jars and had been stored in a freezer duringtransportation and dispensed in a mixture of honey (50%). The patient stored royal jelly and honey mix in a regular refrigerator during its use.

CASE NO. 1: (R.S.)
This is a 52-year-old white female who had two documented mycardial infarctions in February, 1957 and May, 1968, and a subsequent minor attack in December, 1969 followed by congestive heart failure in January, 1974. (All at St. Mary's Hospital, Brooklyn, N.Y.). Other pertinent history was a total hysterectomy in March 1971. She had anginal pain and shortness of breath associated with excessive perspiration. She also complained of pains in the legs. See attached chart (A) for series of cholesterol and triglyceride test results of same patient.

She was picked up because of her initial triglyceride level being 650 mg% in May, 1975 and her cholesterol level was 276 mg% April 23, 1975. August 16, 1974, she started to take one teaspoonful of Royal Jelly twice a day up to October 6, 1975 (51 days). Just before the initiation of Royal Jelly her triglyceride level was 440 mg%: during the interim, her triglyceride level went down to 204 mg% and 5 weeks later 193 mg%. (Normal up to 200 mg%). After cessation of Royal Jelly application, her triglyceride level elevated to 344 mg% on October 10, 1974. (This blood was post prandial specimen).

On December 2, 1974, her triglyceride level was 325 mg% on the fasting blood. Because of the recurrent abnormal changes, the second course of similar amounts of Royal Jelly was given, starting December 10, 1974 and ending December 27, 1974. At the conclusion of the second course, her cholesterol level decreased to 217 mg% and CLM laboratory's results of triglyceride was 315 mg%. (Normal is 150 mg%).

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